Late potentials as predictors of risk after thrombolytic treatment?

or intraoperative endocardial and epicardial mapping"' or indirectly from the body surface by "high-resolution electrocardiography"'7 after myocardial infarction in patients with sustained ventricular tachycardia. These potentials originate from asynchronous electrical activity in bundles of surviving muscle, mostly at the border of a myocardial infarction. These surviving muscle bundles are separated by fibrous tissue that can form isolating boundaries between them.89 The low amplitude of these signals is probably the result of the large amount of connective tissue and few surviving muscle fibres beneath the electrode.8 They are considered as markers for